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GOJAZNOST, DIJABETES I ATEROSKLEROZA: INFLAMACIJA KAO MEHANIZAM POVEZIVANJA

Medicinski glasnik
Jazika rada
Strane
36-38
Ustanova
Odeljenje za gojaznost, metaboličke i reproduktivne poremećaje, Klinika za endokrinologiju, dijabetes i bolesti metabolizma, Klinički centar Srbije, Beograd
Reference
J.R. Sowers, “Obesity as a kardiovascular risk factor”, The Amecican Journal of Medicine, 2003; Vol 115, No 8 pp 37-41.
The World Health Report 2002 , Reducing Risks, Promoting Healthy Life,WHO, Geneva 2002.
N. Aoi, M. Soma, T. Nakayama, et al. “Variable number of tandem repeatof the 5-fl anking region of type C- human natriuretic peptid receptor gene infl uences bood pressure levels in obesity associated hypertension”, Hypertension research, 2004; Vol. 27, N
K.Kosuge, M. Soma, T. Nakayama, “Human uncapling protein 2 and 3 genes are associated with obesity in Japanese”, Endocrine, 2008; Vol. 34, No 1-3, pp 87-95.
K. Strohacker, B.K. Mc Farlin, “Infl uence of obesity, physical inactivity and weight cycling on chronic infl ammation”, Frontiers in Bioscience”,2004, Vol. 2, pp 98-104
V. De Clercq, C. Teylor, P. Zahradka, “Adipose tissue: The link between obesity and cardiovascular disease”,Cardiovascular and Hematological Disorders Drug Targets”, 2007; Vol.8, No3, pp 238-237.
S.K. Fried, D.A. Bunkin, A.s. Greenberg, “Omental and subcutaneous adipose tissue of obese subjects release interleukin 6: depot difference and regulationby glucocorticoid” The Journal of Clinical Endocrinology, 1998; Vol. 83, No 3, pp 843-850
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GOJAZNOST, DIJABETES I ATEROSKLEROZA: INFLAMACIJA KAO MEHANIZAM POVEZIVANJA

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